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Apoptosis(ISBN=9780521626798) 英文原版书籍详细信息
- ISBN:9780521626798
- 作者:暂无作者
- 出版社:暂无出版社
- 出版时间:2004-08
- 页数:202
- 价格:182.80
- 纸张:胶版纸
- 装帧:平装
- 开本:32开
- 语言:未知
- 丛书:暂无丛书
- TAG:暂无
- 豆瓣评分:暂无豆瓣评分
内容简介:
The concept of programmed cell death, or apoptosis, has
exploded into a major scientific field of interest for cell
biologists, oncologists, and many other biomedical researchers.
Apoptosis occurs throughout the lifetime of most multicellular
organisms. During development, for example, the selective death of
cells is vital to remove tissue between the digits to produce
fingers and toes. Apoptosis is also necessary to destroy cells that
represent a threat to the integrity of the organism, for example
cells infected by a virus. In many cancers the genes regulating
apoptosis are defective, producing immortal, continuously
proliferating cells. This book discusses the philosophical and
technical difficulties in defining the moment of death for a cell,
as well as the biological implications and significance of
programmed cell death. Recent developments in the genetic control
and interacting gene networks associated with apoptosis are
presented. The book is written for advanced undergraduate and
postgraduate students, and is highly illustrated to aid
understanding.
书籍目录:
1. Dead or alive
2. How to die
3. What to wear and who clears up the rubbish
4. To reproduce or die
5. The judge, jury, and executioner - the genes that control cell
death
6. Stem cells
7. An in vivo system to study apoptosis: the small intestine
8. Cell death in diverse systems
9. Measuring levels of cell death
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媒体评论
'A compelling read for anyone interested in organismal
development ... This is a timely publication and one that will fill
a much-needed niche on biologist's bookshelves.' Biologist
书籍介绍
This book describes the biological implications and significance of apoptosis. Occurring naturally throughout the lifetime of most multicellular organisms, apoptosis, or programmed cell death, accounts for the continuous turnover of cells in the body. However, the genes regulating apoptosis are defective in many cancers.
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